Scott Michael Blackman, M.D., Ph.D.

Headshot of Scott Michael Blackman
  • Associate Professor of Pediatrics


Cystic Fibrosis, Diabetes, Endocrine Diseases, Endocrinology, Pediatric Endocrinology, Pituitary Disorders, Thyroid more

Research Interests

Use of insulin pumps in infants and toddlers with diabetes; The amount of CFTR function necessary to reduce the risk of CF complications; Determining the biological role of the CFTR protein; Mechanisms of regulating weight gain and nutritional status in CF; The molecular mechanism of CF-related diabetes; Molecular genetics of diabetes and cystic fibrosis more

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Johns Hopkins Pediatrics

Appointment Phone: 443-997-5437
200 N. Wolfe Street
Rubenstein Child Health Building
Baltimore, MD 21287 map
Phone: 410-955-6463 | Fax: 410-955-9773


Dr. Scott M. Blackman is an associate professor of pediatrics at the Johns Hopkins University School of Medicine and attending at the Johns Hopkins Hospital. His areas of clinical expertise include pediatric endocrinology.

Dr. Blackman cares for infants and children with all types of endocrinologic disorders, including diabetes mellitus and disorders of growth, puberty, and the thyroid and pituitary glands. He runs a multidisciplinary clinic for pediatric patients with cystic fibrosis-related diabetes (CFRD).

He received his A.B. at Princeton University and his M.D. and Ph.D. degrees from the Vanderbilt University School of Medicine. He completed his internship and residency in pediatrics and fellowship in pediatric endocrinology at Johns Hopkins. Dr. Blackman has been a faculty member in pediatric endocrinology since completing his fellowship in 2007.

Dr. Blackman’s research interests include identifying genetic variants responsible for endocrine abnormalities in CF including CF-related diabetes, which is seen commonly in teenagers and adults with cystic fibrosis. His long-term research goals include understanding molecular mechanisms of diabetes, such as type 1 and type 2 diabetes as well as CFRD.

He is a recipient of the Gilead Research Scholars Award and has received awards from the Pediatric Endocrine Society and Cystic Fibrosis Foundation. He receives research funding from the NIH and CF Foundation. He serves as a mentor for EnVision, a national program to train endocrinologists in the care of people with CF; he serves on the Program Planning Committee for the North American CF Conference and on the editorial board of the Journal of Cystic Fibrosis.

He serves on study sections for the NIH (NIDDK) and the Cystic Fibrosis Foundation.  He is a member of the Society for Pediatric Research and is a U.S. News Top Doctor. more


  • Associate Professor of Pediatrics

Departments / Divisions

Centers & Institutes



  • MD; Vanderbilt University School of Medicine (2000)


  • Pediatrics; Johns Hopkins University School of Medicine (2004)


  • Pediatric Endocrinology; Johns Hopkins University School of Medicine (2007)

Board Certifications

  • American Board of Pediatrics (Pediatric Endocrinology) (2007)
  • American Board of Pediatrics (Pediatrics) (2004)

Research & Publications

Research Summary

Dr. Blackman’s primary research interest is in how gene variants cause diabetes and other metabolic diseases in people with cystic fibrosis (CF). His projects aim to determine how CFTR and other genes combine to cause cystic fibrosis-related diabetes (CFRD) and other complications of CF.

Clinical Trials

Genetic Modifiers of Cystic Fibrosis Related Diabetes

Selected Publications

View all on PubMed

Lewis C, Blackman SM, Nelson A, Oberdorfer E, Wells D, Dunitz J, Thomas W, Moran A. “Diabetes-related mortality in adults with cystic fibrosis. Role of genotype and sex.” Am J Respir Crit Care Med. 2015 Jan 15;191(2):194-200.

Blackman SM, Raghinaru D, Adi S, Simmons JH, Ebner-Lyon L, Chase HP, Tamborlane WV, Schatz DA, Block JM, Litton JC, Raman V, Foster NC, Kollman CR, DuBose SN, Miller KM, Beck RW, DiMeglio LA. “Insulin pump use in young children in the T1D Exchange clinic registry is associated with lower hemoglobin A1c levels than injection therapy.” Pediatr Diabetes. 2014 Dec;15(8):564-72. doi: 10.1111/pedi.12121. Epub 2014 Feb 4.

Blackman SM, Commander CW, Watson C, Arcara KM, Strug LJ, Stonebraker JR, Wright FA, Rommens JM, Sun L, Pace RG, Norris SA, Durie PR, Drumm ML, Knowles MR, Cutting GR. “Genetic modifiers of cystic fibrosis-related diabetes.” Diabetes. 2013 Oct;62(10):3627-35. doi: 10.2337/db13-0510. Epub 2013 May 13.

Corvol, H, Blackman SM, Boëlle P-Y, Gallins PJ, Pace RG, Stonebraker JR, Accurso FJ, Clement A, Collaco JM, Dang H, Dang AT, Franca A, Gong J, Guillot L, Keenan K, Li W, Lin F, Patrone MV, Raraigh KS, Sun L, Zhou Y, O’Neal WK, Sontag MK, Levy H, Durie PR, Rommens JM, Drumm ML, Wright FA, Strug LJ, Cutting GR, Knowles MR. Genome-wide association meta-analysis identifies five modifier loci of lung disease severity in cystic fibrosis. Nature Comm. 2015 Sep 29;6:8382. doi: 10.1038/ncomms9382. PubMed PMID: 26417704.


Bradley GM*, Blackman SM*, Watson CP, Doshi VK, Cutting GR. Genetic modifiers of nutritional status in cystic fibrosis. Am J Clin Nutr. 2012 Dec;96(6):1299-308. doi: 10.3945/ajcn.112.043406. Epub 2012 Nov 7. PubMed PMID: 23134884,); PubMed Central PMCID: PMC3497925.


Activities & Honors


  • Gilead Research Scholars Award for Cystic Fibrosis, Gilead Sciences, Inc., 2014


  • Pediatric Endocrine Society
  • Endocrine Society
  • American Society of Human Genetics
  • American Diabetes Association
  • Society for Pediatric Research
  • European Cystic Fibrosis Society

Professional Activities

  • Program Planning Committee, North American Cystic Fibrosis Conference
  • Editorial board, Journal of Cystic Fibrosis

Patient Ratings & Comments

The Patient Rating score is an average of all responses to physician related questions on the national CG-CAHPS Medical Practice patient experience survey through Press Ganey. Responses are measured on a scale of 1 to 5, with 5 being the best score. Comments are also gathered from our CG-CAHPS Medical Practice Survey through Press Ganey and displayed in their entirety. Patients are de-identified for confidentiality and patient privacy.

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