| January 2013 |
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Physiology
[Physiology
archives]
1. Gray LW, Peng F,
Molloy SA, Pendyala VS, Muchenditsi A, Muzik O, Lee J, Kaplan JH, Lutsenko
S (2012) urinary copper elevation in a mouse model of Wilson's disease
is a regulated process to specifically decrease the hepatic copper load.
PloS One. 2012; 7(6):e38327.
Key words: Wilson's disease, urinary copper elevation
http://www.ncbi.nlm.nih.gov/pubmed/22802922
2. Hasan NM, Gupta A, Polishchuk E, Yu CH, Polishchuk R, Dmitriev OY,
Lutsenko S. Molecular events initiating exit of a copper-transporting
ATPase ATP7B from the trans-Golgi network. J Biol Chem. 2012 Oct 19;287(43):36041-50.
Key words: copper, ATPase, trans-Golgi network
http://www.ncbi.nlm.nih.gov/pubmed/22898812
3. Tamura Y, Onguka O, Itoh K, Endo T, Iijima M, Claypool SM, Sesaki
H. Phosphatidylethanolamine biosynthesis in mitochondria: phosphatidylserine
(PS) trafficking is independent of a PS decarboxylase and intermembrane
space proteins, Ups1p and Ups2p. J Biol Chem. 2012 Dec 21;287(52):43961-71.
Key words: phosphatidylethanolamine, mitochondria, space proteins
http://www.ncbi.nlm.nih.gov/pubmed/23124206
4. Whited K, Baile MG, Currier P, Claypool SM.
Seven functional classes of Barth syndrome mutation. Hum Mol Genet.
2012 Nov 2. [Epub ahead of print]
Key words: Barth syndrome, mutation
http://www.ncbi.nlm.nih.gov/pubmed/23100323
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