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Adam Scott Wenick, M.D., Ph.D.

Assistant Professor of Ophthalmology
Assistant Professor of Ophthalmology
Specializes In: Adults (18+ years)

Male

Main Location

The Johns Hopkins Hospital

Titles

  • Assistant Professor of Ophthalmology
  • Assistant Professor of Ophthalmology

Centers & Institutes

Departments

  • Ophthalmology - Retinal Vascular Service
  • Surgery at Suburban Hospital

Locations

The Johns Hopkins Hospital

600 N. Wolfe Street
Carnegie 446D1
Baltimore, MD 21287 map

The Johns Hopkins Hospital

Appointment Phone: 410-955-3518

600 N. Wolfe Street
Maumenee
Baltimore, MD 21287 map
Phone: 410-955-3518

Johns Hopkins Wilmer Eye Institute - Bethesda

Appointment Phone: 240-482-1100

7315 Wisconsin Avenue
West Tower, Suite 610
Bethesda, MD 20814 map
Phone: 240-482-1100

Expertise

CMV Retinitis, Diabetic Retinopathy, Macular Degeneration, Ophthalmology, Retina, Medical Diseases, Retina, Surgical Diseases, Retinal Detachment, Retinal Vein Occlusion, Vitreoretinal Diseases and Surgery Service

Biography

Adam S. Wenick, M.D., Ph.D., is an assistant professor of ophthalmology in the retina division at the Wilmer Eye Institute. Dr. Wenick is board certified by the American Board of Ophthalmology and has subspecialty training in vitreoretinal diseases and surgery. His expertise includes medical and surgical treatment of age-related macular degeneration, diabetic retinopathy, retinal vascular occlusions, epiretinal membranes, macular holes, trauma, and retinal tears and detachment.

Dr. Wenick received his M.D. and Ph.D. (in neurodevelopment) degrees from Columbia University. He completed an internship in internal medicine at New York Presbyterian Hospital at Columbia University Medical Center and his ophthalmology residency at the Wilmer Eye Institute at Johns Hopkins University School of Medicine. He completed a fellowship in vitreoretinal disease and surgery at Wilmer before joining the faculty in 2012.

Dr. Wenick’s research interests include the study of the molecular components of the vitreoretinal interface and their role in normal development as well as in the pathogenesis of vitreoretinal disease.

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